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. 2023 Mar 13;56:11. doi: 10.1186/s40659-023-00423-8

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© The Author(s) 2023

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 6 — A–E NFκB1 immunohistochemistry-stained pancreas sections of A; Sham group showed minimal reaction for NFκB1 among acinar cells, B; NAFP group showed extensive positive reaction for NFκB1, C; R-10 group showed positive obvious reaction for NFκB1 among distorted pancreatic acini, D; R-20 group showed moderate positive reaction for NFκB1 in destructed areas, and E; R-30 group showed minimal positive reaction for NFκB1. [Magnification: 200x]. F; The mean area percentage of NFκB1 positive cells (± SD) in the Sham and the experimental groups (n = 6): ^*P < 0.001 vs the Sham group; ^###P < 0.001 vs NAFP group. ^aP < 0.05 vs R-10. ^bP < 0.05 vs R-20