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. 2023 Feb 27;25(3):493–507. doi: 10.1038/s41556-023-01093-0

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Extended Data Fig. 5 — a, Dotplot showing the expression of each selected marker gene per cell type in mouse cerebellum. b, The schematic diagram shows the timeline of Purkinje cell development in both humans and mice. c, UMAP visualization of the developing mouse cerebellum. d, UMAP visualization and marker-based annotation of cell types from the developing human cerebellum. e, Dotplot showing the expression of each selected marker gene per cell type in the human cerebellum. f, The top 20 biological pathways are enriched by Gene-ontology analysis of the SMARCD3-positively correlated genes in the human cerebellum. g, The top 20 diseases are enriched in DisGeNET by analyzing the SMARCD3-positively correlated genes in the human cerebellum. Each dot represents one cell (c, d). Dots with the same color come from the same mouse cerebellum (c) or the same cell type (e). P value from the multiple Fisher test is corrected using two-tailed Benjamini-Hochberg method (f, g).