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. 2023 Mar 17;13:4448. doi: 10.1038/s41598-023-31086-w

Table 4.

The prevalence of virulence-associated genes in K. pneumoniae isolates from different clinical materials.

Genes Source of isolation
Urine n = 39 (%) Blood n = 22 (%) Respiratory tract n = 16 (%) Anus n = 17 (%) Wound n = 13 (%) Other n = 2 (%) Total n = 109 (%)
Adhesins
 fimH 35 (89.7) 19 (86.4) 15 (93.7) 17 (100) 12 (92.3) 2 (100) 100 (91.7)
 mrkD 35 (89.7) 22 (100) 16 (100) 17 (100) 13 (100) 2 (100) 105 (96.3)
Salmochelin
 iroD 4 (10.2) 0 3 (18.7) 0 3 (23.0) 0 10 (9.2)
 iroN 1 (2.6) 2. (9.1) 3 (18.7) 0 2 (15.4) 0 8 (7.3)
Colibactin
 clbA 1 (2.6) 0 0 0 0 0 1 (0.9)
clbB 1 (2.6) 0 0 0 0 0 1 (0.9)
Enterobactin
 entB 39 (100) 22 (100) 16 (100) 17 (100) 13 (100) 2 (100) 109 (100)
Yersiniabactin
 irp-1 36 (92.3) 19 (86.3) 14 (87.5) 17 (100) 9 (69.2) 1 (50) 96 (88.0)
Iron uptake system
 kfu 5 (12.8) 1 (4.5) 1 (6.25) 0 0 0 7 (6.4)
Capsular fucose synthesis
 wcaG 5 (12.8) 3 (13.6) 1 (6.2) 1 (5.9) 2 (15.4) 0 12 (11.0)
Regulator of mucoid phenotype A
 rmpA 2 (5.1) 3 (13.6) 0 0 2 (15.4) 0 7 (6.4)
Capsular types (K)
 K1 9 (23.0) 4 (18.2) 4 (25.0) 0 3 (23.0) 1 (50) 21 (19.2)
 K2 6 (15.4) 6 (27.3) 8 (50.0) 2 (11.7) 3 (23.0) 0 25 (22.9)
 K5 0 0 0 0 0 0 0
 K20 0 0 0 0 0 0 0
 K54 2 (3.7) 0 0 0 2 (9.0) 0 4 (3.7)
 K57 13 (33.3) 5 (22.7) 2 (12.5) 15 (88.3) 4 (30.7) 1 (50) 40 (36.7)