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. 2023 Mar 6;17:1148258. doi: 10.3389/fnins.2023.1148258

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Copyright © 2023 Zulfugarova, Zivari-Ghader, Maharramova, Ahmadian, Eftekhari, Khalilov, Turksoy, Rosić and Selakovic.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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The cellular processes behind the neuroprotective properties of glycyrrhizin. Glycyrrhizin may enhance the integrity of the blood-brain barrier by preventing astrogliosis, neuronal apoptosis, microglia activation, oxidative-induced cellular damage, and mitochondrial dysfunction. These methods enable glycyrrhizin to boost anti-excitotoxicity (for epilepsy treatment), decreasing axonal damage and brain edema (TBI improvement), cognitive and motor function improving (AD and PD treatment), and reducing demyelination (MS treatment). MS, multiple sclerosis; TBI, traumatic brain injury; PD, Parkinson; AD, Alzheimer’s disease.