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. 2023 Mar 21;21:209. doi: 10.1186/s12967-023-04053-2

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© The Author(s) 2023, corrected publication 2023

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 6 — Comparison of proportions of survival-associated genes between NATs and tumors in different TCGA cancer types. A HR and the 95% CI of each gene from SMC-CRC samples are depicted. Only genes with top and bottom 20% HR in NATs (left panel) and tumors (right panel) are shown, respectively. Significant survival-associated genes (P < 0.05; Cox regression analysis) are colored blue for NAT and red for tumor. B Same as (A) but for TCGA-COAD samples. Note that 79 outlier genes with HR > 100 or HR < 0.01 in either NATs or tumors were removed. C–G Pie charts for the significant survival-associated genes (P-value < 0.05; Cox regression analysis) in (C) SMC-CRC, D TCGA-COAD, E TCGA-BRCA, F TCGA-LIHC, and G TCGA-LUAD are shown