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. 2023 Mar 8;615(7953):652–659. doi: 10.1038/s41586-023-05791-5

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Extended Data Fig. 5 — Variants (SNPs and short InDels) resulting in premature termination codons (PTCs) identified from SPET data. a, Distribution of allele counts including missing genotype calls. b, Distribution of allele frequencies excluding missing genotype calls. c, Summary of resistance gene analogues (RGAs) identified in the Hedin/2 genome annotation. d, Distribution of allele frequencies of variants resulting in PTC in RGAs. e, Permutation test for over-representation of PTCs in RGAs.