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. 2023 Jan 24;482(3):507–516. doi: 10.1007/s00428-022-03484-4

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© The Author(s) 2023

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — Representative examples of pancreatic ACTH-secreting neuroendocrine neoplasms. A + B This case (case 1 in Table 1 with EWSR1::BEND2 fusion) showed features of NETG3 with extensive foci of necrosis. C Liver metastasis from pancreatic NETG3 with KMT2A::BCOR fusion (case 2). D Primary pancreatic NETG3 (case 10 in Table 1 with TFG::ADGRG7 fusion). All tumors expressed synaptophysin (E), chromogranin-A (not shown), somatostatin receptor 2A (F), and ACTH (G)