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. 2023 Mar 23;22:15330338231165125. doi: 10.1177/15330338231165125

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© The Author(s) 2023

This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (https://creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access page (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Figure 3. — Representative images of tumors with different gene mutations. (A) Gross morphology, nodule phenotypes, and hematoxylin and eosin staining of tumors with Tp53 + Pten, Tp53 + Pten + Nf2, and Tp53 + Pten + Tsc2 mutations. (B) Gross morphology, nodule phenotypes, and hematoxylin and eosin staining of metastatic tumors with Tp53 + Pten and Tp53 + Pten + Tsc2 mutations. (C) Immunohistochemical analyses of Tp53, Pten, Nf2, and Yap1. Staining of other stromal cells in the liver served as a positive control.