Table 1.
Type of Pain | Aetiology | Clinical Considerations |
---|---|---|
Neuropathic pain/ Radiculopathy |
Caused by a lesion or disease of the somatosensory system, including peripheral fibres and central neurons [15], not always associated with a response to external stimulus, pain signalling pathways are damaged. | Ca. 30% of neuropathic pain is caused by diabetes, common in cancer survivors and neurodegenerative diseases. Lumbar and cervical painful radiculopathies often cause neuropathic pain [15]. Sleep disturbances, anxiety and depression are frequent and severe in patients with neuropathic pain [16]. |
Blocking of conduction along sensory and motor axons results in loss of nerve function [17,18], compressed or inflamed nerve route leads to loss of function. | Causing pain, loss of sensation, and motor function depending on the severity, lumbosacral radiculopathy is very common [19], e.g., herniated disc with resultant nerve root compression or spondylosis [19]. | |
Radicular pain | Pain radiates from an inflamed or compressed nerve root, not from stimulation of peripheral nerve endings, is not nociceptive pain [17]. | Irritation of a sensitized nerve root, usually of a prolapsed intervertebral disc, pain is typically lancinating, shooting down the leg [18]. |
Nociceptive pain—2 types | Resultant from tissue damage by physical, chemical or traumatic events, injuries or infections [4], pain signalling pathways are intact. | |
|
Peripheral areas of the body | Triggered by an acute injury or chronic disease. |
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Internal organs | Frequently associated with nausea, vomiting, nervousness. |