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. 2023 Apr 4;24:172. doi: 10.1186/s12864-023-09280-x

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© The Author(s) 2023

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — Differential splicing analysis using DEXSeq. A Number of genes with differential exon usage (DEU) and differential expressed genes (DEG) in each mouse model. Common infers gene overlap between DEG and DEU genes. B Significantly enriched KEGG pathways in the set of differential exon usage (DEU) and differentially expressed genes (DEG) in the 12 months old Trem2*R47H mice (p < 0.05). C Cell type enrichment analysis in the gene set with differential exon usage (DEU) and differentially expressed genes (DEG) in each mouse model using Fisher exact test. Significant cell type enrichment (p < 0.1) are shown by solid dots