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. 2023 Mar 22;5(2):fcad080. doi: 10.1093/braincomms/fcad080

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© The Author(s) 2023. Published by Oxford University Press on behalf of the Guarantors of Brain.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (https://creativecommons.org/licenses/by/4.0/), which permits unrestricted reuse, distribution, and reproduction in any medium, provided the original work is properly cited.

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Correlation analyses. In the principal component and path analyses, we found two paths, one from extravascular blood volume component (blood_component) to delayed cerebral ischaemia component (DCI_component) with spreading depolarization component (SD_component) as mediator variable, and one from IVH to DCI_component with DSA component as mediator variable. (A) Three strongest correlations between individual variables from the three variable groups involved in the path from blood_component to SD_component to DCI_component. (B) Three strongest correlations between individual variables from the three variable groups involved in the path from IVH to DSA_component to DCI_component. (Ci) The electrode strip was typically located on the cortex of the territory of the MCA. However, there was no correlation between angiographic vasospasm in the M1 segment (or M2 segment, see Supplementary Table 1) of the MCA (DSA_M1) with the SD variables [shown here is the PTDDD_delayed (peak value of a recording day for the total (cumulative) SD-induced depression durations during the delayed period)]. (Cii) In contrast, the TCD-determined peak mean blood flow velocity of the MCA (mbfv_MCA) correlated with DSA_M1. blood_convex = subarachnoid blood volume on the cerebral convexity; DCI_ACA = delayed infarct volume in the territory of the anterior cerebral artery (ACA); DCI_MCA = delayed infarct volume in the territory of the MCA; DSA_A2 = DSA score of the A2 segment of the ACA.