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. 2023 Apr 8;14(4):258. doi: 10.1038/s41419-023-05776-2

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — A The efficient overexpression and suppression of PRPF19 were confirmed by WB in the indicated stable cell lines. B, C Transwell assays showed the migration and invasion ability of HCT116 and DLD1 cells with PRPF19 overexpression or knockdown (left panel), and cell quantification was shown (right panel). D, E Representative images showed the wound healing capability of HCT116 and DLD1 cells with PRPF19 overexpression or knockdown. ***p < 0.001, **p < 0.01, *p < 0.05 based on Student’s t test. These experiments were repeated at least three times. Error bars, mean ± SD.