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. 2023 Mar 28;14:1149478. doi: 10.3389/fphar.2023.1149478

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Copyright © 2023 Zhang, Cao, Zuo, Cheng, Liu, Xia, Ren, Deng, Wang and Lu.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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BA inhibited the PI3K/Akt signaling pathway. (A,B) The PI3K/Akt pathway related protein expression levels of HCT116 (A) and CT26 (B) cells treated with BA were measured by western blot, and β-tubulin served as a loading control. (C,D) The cell inhibition rate of HCT116 (C) and CT26 (D) cells were treated with BA and/or LY294002 for 48 h (E,F) The PI3K/Akt pathway related protein expression levels of HCT116 (E) and CT26 (F) cells treated with BA and/or 2 LY294002 (20 μM) were measured by western blot, and β-tubulin or β-actin served as a loading control. (G,H) Western blotting intensity and standardized statistics of PI3K and Akt in HCT116 (G) and CT26 (H) cells with or without BA in CETSA test. *p < 0.05, **p < 0.01, ***p < 0.001, compared to the control group.