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. 2023 Apr 14;132(8):970–992. doi: 10.1161/CIRCRESAHA.123.321752

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© 2023 The Authors.

Circulation Research is published on behalf of the American Heart Association, Inc., by Wolters Kluwer Health, Inc. This is an open access article under the terms of the Creative Commons Attribution Non-Commercial-NoDerivs License, which permits use, distribution, and reproduction in any medium, provided that the original work is properly cited, the use is noncommercial, and no modifications or adaptations are made.

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Figure 5. — Endothelial phenotype in chronic kidney disease (CKD) conditions in relation to vasoreactivity. Endothelial phenotype in CKD conditions in terms of vasodilation/vasoconstriction. For more information, see text. eNOS indicates endothelial nitric oxide synthase; ER, endoplasmic reticulum; HCO₃⁻, bicarbonate; HDL, high-density lipoprotein; H₂O₂, hydrogen peroxide; LOX-1, lectin-type oxidized low-density lipoprotein receptor 1; NADPH, nicotinamide adenine dinucleotide phosphate; NO, nitric oxide; NOX, NADPH oxidase; O₂, oxygen; O₂^*−, superoxide; SDMA, symmetric dimethylarginine; TLR2, toll-like receptor 2; and TNFɑ, tumor necrosis factor ɑ.