Table 2.
Clinical features and key information for the cases presented.
| Cases | Patient characteristics | Sex | Age (years) | Etiology | NCV/EMG | K+ level | Neurological examination | Family history of similar disease | Possible triggers |
|---|---|---|---|---|---|---|---|---|---|
| Katabi A et al.[1] (HypoKPP) | Bilateral leg and left arm weakness | M | 40 | NA | NA | 1.9 mmol/L (normal range: 3.5–5.0mmol/L) | NA | No | History of recent exercise |
| Negrotto L et al.[2] (secondary low serum potassium) | An isolated mild right brachial paresis, and extend to upper and lower limbs weakness on admission | F | 51 | Renal tubular acidosis | Normal | 2.2 mEq/L (normal range: 3.5–5.0 mEq/L). | 1.Onset stage: NA 2.On admission: predominantly proximal grade 2/5 |
NA | NA |
| Left-hand weakness | M | 47 | Primary hyperaldosteronism | Normal | 1.8 mEq/L (normal range: 3.5–5.0 mEq/L). | Weakness of left wrist and finger extensors (grade 2/5 and 3/5, respectively) and “mild weakness of left interosseous muscles” |
NA | NA | |
| Lu YT et al.[3] (suspected HypoKPP) | Right arm and leg weakness | M | 52 | Subclinical corticospinal tract damage hypothesis | NA | 1.8 mEq/L (normal range: 3.0–4.8 mEq/L) | Grade 2/5 | NA | NA |
| Chui C et al.[4] (secondary low serum potassium) | “Could not extend or elevate her left thumb” | F | 48 | Adrenal adenoma | Normal | 1.9 mEq/L (normal e range: 3.4–5.0) | NA | No | Frequent exercise in her job |
NA = not available; F = female; M = male; NCV = nerve conduction velocity; EMG = electromyography.