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. 2023 Mar 9;97(5):1285–1298. doi: 10.1007/s00204-023-03455-x

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© The Author(s) 2023

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — Slides of renal, brain and gastric tissue stained with eosin hematoxylin of control and high dose subjects. Magnification × 200. A Renal tissue control × 200. RG Renal glomerulus. Normal shape and number of renal glomeruli with normal cellularity level. B Brain tissue control × 200, AC astrocytes, GC glial cells. Normal number of glial cells and astrocytes. C Gastric tissue control × 200 GM Gastric mucosa, GG Gastric glands, MM Muscularis mucosae with normal architecture with no distortion of the mucosal integrity and no signs of inflammation. D Renal tissue high dose (HD) × 200. TC Tubular cells. Severe numerical reduction in cellularity with intense growth in tubular cell aggregates forming renal tubules molds. E Brain tissue high dose (HD) × 200, AC astrocytes, GC glial cells. Severe numerical reduction of glial and astrocyte cells. F Gastric tissue high dose (HD) × 200 GM Gastric mucosa, GG Gastric glands, GC granulocytes, MM Muscularis mucosae with severe distortion of mucosal architecture and necrotic lesions of mucosal foci along with mucosal degeneration