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. 2022 Nov 17;72(5):1327–1335. doi: 10.1007/s00262-022-03327-w

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — STING expression in non-tumor cells differentially regulates response to ICB in MC38 and B16F10 models. A Tumor growth in the MC38 tumor model with anti-PD1. B Effect of anti-PD1, anti-CTLA-4, or combination treatment on tumor growth in B16F10 model. C MC38 or D B16F10 tumors implanted in WT or STING KO (gt/gt) mice and treated as indicated. (n = 5–10 animals/group)