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. 2023 Apr 19;39(9):1439–1453. doi: 10.1007/s12264-023-01060-7

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© Center for Excellence in Brain Science and Intelligence Technology, Chinese Academy of Sciences 2023. Springer Nature or its licensor (e.g. a society or other partner) holds exclusive rights to this article under a publishing agreement with the author(s) or other rightsholder(s); author self-archiving of the accepted manuscript version of this article is solely governed by the terms of such publishing agreement and applicable law.

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Fig. 3 — Dual-target anti-stroke drug ZL006-05. ZL006-05 blocks the nNOS-PSD-95 interaction while selectively potentiating ɑ2-containing GABA_ARs and thereby inhibits NO overproduction and reduces excitability, consequently preventing stroke damage and relieving chronic pain. GABA_ARs, GABA type A receptors; NMDAR, N-methyl-D-aspartate receptor.