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. 2023 Apr 20;24:25. doi: 10.1186/s40360-023-00660-3

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© The Author(s) 2023

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Effects of hypoxia and reperfusion on the contractile function of cardiomyocytes. An example of the recording obtained during the experiment. The trabeculae's contraction amplitudes are shown for remifentanil (above) and the control (below). A profound decrease in trabeculae contraction results from ischemia. At the end of 60 min of ischemia, oxygen is again applied (reperfusion), and opioid is added to the study probe. Higher contraction amplitudes are noted for the remifentanil probe