Table 1.

Demographics, imaging features, and clinical information.

	MLH (n = 7)	Non CAA (n = 4)	CAA (n = 3)
Age, mean (SD)	69 (8.7)	69 (12.1)	69 (2.8)
Female sex, n (%)	2 (29)	1 (25)	1 (33)
Fazekas score, median (IQR)
PVM	3 (2.5, 3)	3 (2.75, 3)	3 (2.5, 3)
DWM	2 (1.5, 2)	2 (1.75, 2)	2 (1.5, 2)
White matter pattern, n (%)
Subcortical	2 (29)	0 (0)	2 (66)
Peri BG	0 (0)	0 (0)	0 (0)
Frontal	1 (14)	1 (25)	0 (0)
Posterior	6 (86)	3 (75)	3 (100)
Lobar lacunes	0 (0)	0 (0)	0 (0)
cSS, n (%)	4 (51)	2 (50)	2 (66)
Ratio superficial/deep CMB, median (IQR)	9.33 (2.67, 17)	6.59 (3.26, 9.75)	23 (11.75, 93.75)
Ischemia, n (%)
Chronic	2 (29)	2 (13)	0 (0)
Acute	4 (51)	2 (50)	2 (66)
EPVS, n (%)
BG	1 (14)	1 (25)	0 (0)
CS	2 (29)	1 (25)	1 (33)
FLP, n (%)	4 (51)	2 (50)	2 (66)
SAH, n (%)	4 (51)	1 (25)	3 (100)
Hemorrhage volume, mean (SD)	70.7 (31.6)	67.0 (41.0)	75.7 (20.2)
Intraventricular extension, n (%)	0 (0)	0 (0)	0 (0)
Antithrombotics, n (%)	3 (43)	1 (25)	2 (66)
Statin use, n (%)	3 (43)	1 (25)	2 (66)
Early ICH related death, n (%)	2 (29)	0 (0)	2 (66)
Elevated creatinine, n (%)	1 (14)	0 (0)	1 (33)
Elevated LDL, n (%)	0 (0)	0 (0)	0 (0)
Arterial hypertension, n (%)	4 (51)	2 (50)	2 (66)
Diabetes mellitus, n (%)	0 (0)	0 (0)	0 (0)
Dyslipidemia, n (%)	0 (0)	0 (0)	0 (0)

No statistically significant differences between CAA and non-CAA patients could be detected.

CAA, cerebral amyloid angiopathy, MLH, mixed location hemorrhages/cerebral microbleeds, PVWM, periventricular white matter, DWM, deep white matter, cSS, cortical superficial siderosis, SAH, subarachnoid hemorrhage, IQR, interquartile range, SD, standard deviation, BG, basal ganglia, EPVS, enlarged perivascular spaces, CS, centrum semiovale, CMB, cerebral microbleeds.