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. 2023 Mar 25;16(4):863–879. doi: 10.1111/eva.13542

FIGURE 3.

FIGURE 3

Genomic responses to abamectin selection. (a) Averaged genome‐wide allele frequency differences between abamectin‐selected and unselected replicates in aBSA (blue line) and gBSA (orange line). QTL associated with abamectin resistance (QTL 1–4) shows peaks that deviate from the genome‐wide average. Dashed lines delineate the statistical threshold for QTL detection (FDR < 0.05). (a–f) Genomic positions are denoted on the x‐axis, with the three pseudochromosome (Chr1‐3) configuration of T. urticae indicated by alternating shading. (b–d) Candidate genes within 500 kb genomic brackets around QTL peaks. Triangles positioned along the top and bottom boundaries of each plot represent the top genomic window, averaged for each BSA experiment: aBSA (blue), gBSA (orange). The orientation of the gene models is indicated by “+” or “−” for forward and reverse strands respectively. Putative candidate genes within the QTL peaks are highlighted in red. (b) QTL peak 1: glutamate‐gated chloride channel 2 (TuGluCl2: tetur08g04990); (c) QTL peak 2: glutamate‐gated chloride channel 3 (TuGluCl3: tetur10g03090); (d‐e) QTL peak 3: glutamate‐gated chloride channel 1 (TuGluCl1: tetur02g04080) and DEAD/DEAH box DNA helicases (tetur02g04060, tetur02g04410) and (f) QTL peak 4: multiple chemosensory receptors (Table S7).