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. 2023 Apr 12;15(4):950. doi: 10.3390/v15040950

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© 2023 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).

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Longitudinal analysis of plasma viral load and cell-associated HIV-1 DNA. (A) Plasma HIV-1 viral load was monitored longitudinally by real-time PCR. (B) Levels of cell-associated HIV-1 DNA were monitored longitudinally by real-time PCR. (C) Kaplan–Meier curve depicts the percent of animals with undetectable plasma viral load following exposure. Data in (A,B) are expressed as mean± SEM. Statistical significance was determined using a two-sided Kruskal–Wallis test in (A,B), and a Mantel–Cox test in (C). p values between WT and MUT1, WT and MUT2 in (C) are <0.0001 and 0.0033, respectively.

Longitudinal analysis of plasma viral load and cell-associated HIV-1 DNA. (A) Plasma HIV-1 viral load was monitored longitudinally by real-time PCR. (B) Levels of cell-associated HIV-1 DNA were monitored longitudinally by real-time PCR. (C) Kaplan–Meier curve depicts the percent of animals with undetectable plasma viral load following exposure. Data in (A,B) are expressed as mean± SEM. Statistical significance was determined using a two-sided Kruskal–Wallis test in (A,B), and a Mantel–Cox test in (C). p values between WT and MUT1, WT and MUT2 in (C) are <0.0001 and 0.0033, respectively.