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. 2023 May 3;16:93. doi: 10.1186/s12920-023-01515-w

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© The Author(s) 2023

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 8 — The relationship and function of ST6GALNAC3 with EMT and macrophage infiltration. The relationship between LMAGs and macrophage infiltration (A, B). The relationship between LMAGs and EMT (C). In the CCLE database, ST6GALNAC3 is significantly positively correlated with EMT markers (ROR2 and TAGLN) (D-E). The expression of ST6GALNAC3 in various gastric cancer cell lines (F). ROR2 and TAGLN decreased significantly with the knockdown of ST6GALNAC3 though PCR (G)