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. 2023 May 6;24:355. doi: 10.1186/s12891-023-06461-3

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© The Author(s) 2023

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — Histopathology of biopsied biceps brachii muscle. A Hematoxylin and eosin (HE) staining revealed many degenerating or atrophied muscle fibers (arrows) but no necrotic fibers. B Modified Gomori–Trichrome (GT) staining revealed nemaline rods (arrows) in some fibers and abnormal cytoplasmic agglutination (arrow heads) within atrophied muscle fibers. C Nicotinamide adenine dinucleotide tetrazolium reductase (NADH-TR) staining revealed a lobulated appearance (arrows) and focally increased or decreased oxidative enzyme activity. D Electron microscopy (EM) indicated that nemaline bodies (arrows) were of the same electron density as the Z line, with some bodies being physically connected to the Z line (arrow heads)