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. 2023 May 6;21:307. doi: 10.1186/s12967-023-04141-3

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© The Author(s) 2023

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 7 — Sp1 promoted the tumorigenesis of cervical cancer. Sp1 is involved in promoting cell proliferation, migration, invasion, and tumour growth and inhibiting cell apoptosis in cervical cancer. Sp1 boosts the tumorigenesis of cervical cancer by reducing aerobic glycolysis and promoting mitochondrial remodelling by rebuilding the balance between mitofusin and mitofission, and then it promotes cell growth and inhibits tumour cell apoptosis in cervical cancer