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. 2023 May 8;23:415. doi: 10.1186/s12885-023-10894-3

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© The Author(s) 2023

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — Proteogenomic regulation of ferroptosis regulators. (A) Barplot showing the SNV class in CPTAC-GBM and TCGA-GBM cohorts, respectively. (B) Top 10 mutated genes in CPTAC-GBM and TCGA-GBM cohorts, respectively. (C) Oncoplots showing the five common mutated genes in both CPTAC-GBM and TCGA-GBM cohorts. (D) Heatmap showing the differential expression of mRNA, protein, and phosphoprotein between mutated and wild samples. (E) Heatmap showing the effect of EGFR, IDH1, PIK3CA, RB1, and TP53 mutations on protein abundances of ferroptosis regulators