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. 2023 May 13;22:112. doi: 10.1186/s12933-023-01833-4

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© The Author(s) 2023

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 5 — Exposure of HCAECs to DVEs promotes increased surface expression of ICAM-1. A Histograms of cell surface expression of ICAM-1 expression following 6 h of treatment. B Fluorescence intensity of ICAM-1 antibody relative to the isotype control (mean ± SEM, n = 3). C Percentage of HCAECs with positive expression of ICAM-1 (mean ± SEM, n = 3). D Histograms of cell surface expression of ICAM-1 expression following 24 h of treatment. E Fluorescence intensity of ICAM-1 antibody relative to the isotype control following 24 h of treatment (mean ± SEM, n = 3). F Percentage of HCAECs with positive expression of ICAM-1 following 24 h of treatment (mean ± SEM, n = 3). *p < 0.05, **p < 0.01, ***p < 0.001