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. 2023 May 2;13:1144184. doi: 10.3389/fonc.2023.1144184

Table 4.

The function of histone methyltransferases in GBM.

Enzyme Associated role in GBM Cell lines/model system used References
EZH2 Overexpressed in GBM. Increases cell cycle progression, invasiveness, tumorigenicity, and tumor growth. Modulates metabolism, differentiation, and immune signaling. A172, BCRC 60380, BCRC 60163, GL261, H4, LN18, LN229, N33, T98G, U87MG, U251, patient-derived GSCs, patient-derived neurospheres, nude mouse model, syngeneic mouse model (1214, 19, 32, 127, 128, 130, 132, 134145, 176186)
EHMT2 Promotes proliferation, migration, and invasion. Reduces differentiation, apoptosis, and autophagy. A172, LN18, LN229, U87MG, U251MG, patient derived GBM cell cultures, patient derived GSCs, nude mouse model (148152, 187)
Suv39H1 Overexpressed in GBM. Promotes proliferation, migration, and colony formation. T98G, U87MG (155, 156)
SETDB1 Overexpressed in GBM. Promotes proliferation, migration, and colony formation. T98G, U87MG (155, 156)
PRMT1 Decreases GBM cell viability in a counter-regulatory fashion to EHMT2 A172, U87MG (152)
PRMT3 Overexpressed in GBM. Regulates glycolysis and inhibition increases apoptosis, inhibitors cell cycle progression, decreases tumor growth U87, U251, patient derived GSCs, nude mouse flank model (163)
PRMT5 Overexpressed in GBM. Promotes colony formation, migration, and cell cycle progression. LN229, U87EGFRvIII, patient derived GBM cell cultures, patient derived neurospheres, patient derived GSCs, zebrafish GBM model, nude mouse model (161, 164, 166, 167)
PRMT6 Overexpressed in GBM. Promotes cell proliferation. T98G, U87, patient derived GBM cells, nude mouse model (162)
MLL1 Promotes self-renewal and tumor formation. Patient derived GSCs, nude mouse model (168)
MLL5 Promotes self-renewal Patient derived GBM cell lines, nude mouse model (169)
SMYD3 Promotes cell proliferation and tumorigenicity HEB, LN18, T98G, U87, U373, nude mouse flank model (188)
DPY30 Promotes cell viability through regulation of hypoxia and angiogenesis Patient derived GBM cell lines, patient derived GSCs, nude mouse model (170, 189)
SETD2 Antiproliferative effects are neutralized in GBM through mutation or EGFR suppression Patient derived GBM cell lines, patient derived GSCs, nude mouse model (171, 172)
KMT5B/5C Dysregulation implicated in gliomagenesis Baboon and mouse-derived NSCs (173)

A summary of the functions of individual histone methytransferase enzymes in GBM pathophysiology, the associated histone post-translational modification, and the corresponding model system(s) used and reference to the original publication(s).