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. 2023 May 18;23:160. doi: 10.1186/s12906-023-03975-0

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© The Author(s) 2023

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 5 — The cantharidin treatment affects chemokine related immune response. (A, B) GO functional annotation shows that cantharidin treatment is positively associated with chemokine biosynthetic and metabolic processes based on GSEA analysis. (C) Venn diagram shows that the differently expressed genes of chemokine and chemokine receptor in the cantharidin treated HepG2 cell. (D, E) The heatmap shows the expression of chemokine (D), and chemokine receptor (E) in the differently expressed genes of cantharidin treated HepG2 cell. (F) The heatmap shows the association of chemokine and chemokine receptor with EZH2 in LIHC, * P < 0.05 was regarded as statistically significant