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. 2023 May 4;9(5):720–732. doi: 10.1038/s41477-023-01405-0

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Extended Data Fig. 5 — (a-b) Normal (NS1⁺/ns1, ns2; a) and wox3a wox3b double mutant plants (b) are phenotypically equivalent (n = 3). Scale bars = 5 cm. (c-d) Effects of WOX3A dosage in a ns1 ns2 background. Transverse cross-sections of the shoot apex of the indicated genotypes stained with toluidine blue. The mediolateral extent of leaves is reduced in ns1 ns2 WOX3A⁺/wox3a mutants (c) relative to ns1 ns2 mutants (compare to Fig. 1h) but less so than in the triple wox3 mutant (d) indicating a dosage effect of WOX3A on phenotypic severity (n = 3). Scale bars = 250 μm.