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. 2023 May 23;213:130–138. doi: 10.1016/j.biochi.2023.05.014

Fig. 1.

Fig. 1

Interactions between oncogenic factors induced by SARS-CoV-2 infection. For details of the interactions see text. RAAS, renin-angiotensin-aldosterone system; Ang, angiotensin; AT1R, angiotensin 1 receptor; AT2R, angiotensin 1 receptor; MAS1R, MAS1 receptor; MAPK, mitogen-activated protein kinase; ERK, extracellular signal-regulated kinase; TGF-β, Transforming growth factor beta; VEGFA, Vascular endothelial growth factor A; HIF-1α, Hypoxia-inducible factor 1-alpha; STAT, signal transducer and activator of transcription; Pim1, Proviral integration site for Moloney murine leukemia virus-1; Pim2, Proviral integration site for Moloney murine leukemia virus-2; C-MYC, Cellular Myelocytomatosis Oncogene; NF-κB, nuclear factor-kappa B; IL-6, Interleukin 6; ROS, Reactive oxygen species; IL-17, Interleukin 17; IL-1β, Interleukin 1β; TNF, Tumor necrosis factor; HSP-27, heat shock protein 27; DDX10, DEAD-box helicase 10; NUP-98, nucleoporin-98; PRB, retinoblastoma protein; GNB-1, Guanine nucleotide-binding protein subunit beta-1; ETC, electron transport chain; HMOX1, heme oxygenase-1; SIRT5, Sirtuin 5; NSD2, Nuclear receptor binding SET domain protein 2; HDAC2, Histone deacetylase 2; NUP214, nucleoporin 214; AKAP9, A Kinase Anchor Protein 9; BRD2, Bromodomain-containing protein 2; BRD4, Bromodomain-containing protein 4; LARP1, La-related Protein 1.