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. 2023 May 23;9:173. doi: 10.1038/s41420-023-01470-6

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — Immunoblotting for TP53 and P21 proteins for controls (a), Swyer and Swyer-GCT (b), CAIS and CAIS-GCT (c), Turner (d) and Klinefelter (e) individuals. (f) Relative quantification full-length (TP53) and depleted isoform (deltaTP53) of TP53 in DSD-groups. Immunoblotting (g) and relative quantification normalized to α-actinin (h) for TGCT-group. Relative quantification of P21 protein in DSD (i) and TGCT (j) groups. Quantification of relative length of telomeres in DSD (k) and TGCT (l) samples.