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. 2023 May 9;11:1181764. doi: 10.3389/fcell.2023.1181764

TABLE 1.

Summary of H3 methylation master regulators considered as potential therapeutic targets in PCa.

Enzyme type Enzyme name PTMs Function in PCa References
HMTs SMYD3 H3K4me2/3 Promotes cell proliferation and migration Vieira et al. (2015)
SUV39H1 H3K9me3 Enhance prostate cancer cell and migration Baratchian et al. (2022)
G9a H3K9me2 Contributes to the development of PCa Chen et al. (2012)
Jones et al. (2021)
EZH2 H3K27me2/3 Crucial key driver of prostate cancer development Yang and Yu (2013)
Abida et al. (2019)
NSD2 H3K36me2 Promotes prostate cancer tumorigenesis and progression. Ezponda et al. (2013)
DOT1L H3K79me2/3 Is necessary for the viability of AR+ PCa Vatapalli et al. (2020)
PRMT4 (CARM1) H3R17me2a Overexpressed in prostate cancer. Associated with tumorigenesis and cancer progression Hong et al. (2004)
PRMT5 H3R8me2a Promotes development of CRPC Beketova et al. (2022)
PRMT6 H3R2/17/42me2a Overexpressed in prostate cancer Vieira et al. (2014)
HDMs KDM5B (JARID1B) H3K4me1/2/3 AR coactivator. Upregulated in PCa Khan et al. (2019)
KDM5C (JARID1C) H3K4me2/3 Controls the proliferation of prostate cancer cells; might represent a novel therapeutic target. Stein et al. (2014)
JMJD2A (KDM2A) H3K9me3 Promotes prostate cancer initiation. Associate with PCa aggressiveness. Kim et al. (2016)
H3K36me3
JMJD3 (KDM6B) H3K27me3 Overexpressed in metastatic prostate cancer Xiang et al. (2007)
JMJD6 H3R2me2 Promotes castration resistant disease Paschalis et al. (2021)
PADI2 H3R26me Promotes AR signaling activation and progression of CRPC Wang et al. (2017)