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. 2023 May 27;8:220. doi: 10.1038/s41392-023-01439-y

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — PTMs in metabolic diseases. An increasing number of people are suffering from metabolic diseases, such as fatty liver, diabetes, obesity, hyperlipidemia and atherosclerosis. The liver, pancreas, adipose tissue, blood vessels, and heart are the main affected organs. Numerous proteins and PTMs (such as phosphorylation, acetylation, methylation, ubiquitination, etc.) are involved in the normal biology of these organs and the whole body. Abnormal PTMs thus are involved in the progression of these metabolic diseases and can be therapeutically targeted. The figure is generated with BioRender (https://biorender.com)