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. 2023 May 27;23:330. doi: 10.1186/s12903-023-03034-7

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 4 — Anti-angiogenic drugs significantly inhibit the clonogenic and proliferative abilities of gingival fibroblasts than anti-resorptive drugs. A Crystal violet staining and 40 × microscope observation of 6-well plates with gingival fibroblasts seeded to observe the colony formation ability. B Statistical analysis of colony formation numbers of gingival fibroblasts post different drug treatments. C Statistical analysis of gingival fibroblasts post different drug treatments using CCK-8 assay. ANOVA was performed. *P < 0.05; **P < 0.01. Scale bars in A (left panel),1 mm; A (right panel), 100 µm