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. Author manuscript; available in PMC: 2024 Jul 15.
Published in final edited form as: Life Sci. 2023 Apr 28;325:121738. doi: 10.1016/j.lfs.2023.121738

Fig. 2.

Fig. 2.

Representative recordings of K+ currents in capsaicin-sensitive bladder afferent neurons from mice. A: Recordings show superimposed outward K+ currents evoked by depolarizing voltage pulses to 0 mV from holding potentials (HPs) of −120 and −40 mV. Sustained delayed rectifier-type K+ (sustained KDR) currents were evoked by depolarization from −40 mV HP. B: Recordings slow decaying A-type K+ (slow KA) currents were obtained by subtracting K+ currents evoked by depolarization to 0 mV from HPs of −40 and −120 mV. SI; spinal intact group, SCI; spinal cord injury group, SCI + p38 MAPK group; spinal cord injury treated with p38 MAPK inhibitor (0.51 μg/h, i.t.) for two weeks. The number of neurons/mice per group is 24/9; SI, 24/9; SCI and 18/7; SCI + p38 MAPK inhibitor, respectively.