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Frontiers in Immunology logoLink to Frontiers in Immunology
. 2023 May 19;14:1216456. doi: 10.3389/fimmu.2023.1216456

Corrigendum: Crosstalk between cGAS-STING pathway and autophagy in cancer immunity

Qijun Lu 1, Yukun Chen 2, Jianwen Li 1, Feng Zhu 3, Zhan Zheng 1,*
PMCID: PMC10236312  PMID: 37275881

In the published article, there was an error in Figure 1 as published. The word ‘immunity’ is not capitalized, keeping the format consistent. The corrected Figure 1 and its caption “ Figure 1 . appear below.

Figure 1.

Figure 1

Schematic illustration of the crosstalk between the cGAS-STING pathway and autophagy in cancer immunity. The figure was created with BioRender.

In the published article, there was an error in Figure 4 as published. The icon (2) in Figure 4 is missing. The corrected Figure 4 and its caption “ Figure 4 appear below.

Figure 4.

Figure 4

The upstream and downstream of the cGAS-STING pathway, including STING proteins, trigger autophagy by the following roughly divided mechanisms: cGAS binds to dsDNA to form liquid-phase condensates. (1) cGAS interacts with Beclin1 and triggers canonical autophagy; (2) cGAS binds to LC3 to induce non-canonical autophagy; (3) cGAS binds to dsDNA and recruits ATG, LC3, and P62 to participate in canonical autophagy; (4) STING leads to ER stress, mTOR inactivation, and coordinates autophagy; (5) STING stimulates RAB22A-mediated non-canonical autophagy derived from the ER; (6) STING recruits ATG16L1 to lipidated LC3, induces non-canonical autophagy. The figure was created with BioRender.

In the published article, there was an error in the author list, and author Yukun Chen was erroneously denoted as co-first author. The corrected author list appears below.

Qijun Lu1, Yukun Chen2, Jianwen Li1, Feng Zhu3 and Zhan Zheng1*

The authors apologize for these errors and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.

Publisher’s note

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