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. 2023 Jun 3;23:501. doi: 10.1186/s12885-023-10983-3

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 7 — Transient knockdown of HSPB1 affects the cell epithelial-mesenchymal transition (EMT) process. A Vimentin, N-cadherin, and E-cadherin expression levels after siHSPB1-treatment as measured using western blotting. All assays were performed in triplicate. The blots were cut prior to hybridisation with antibodies during blotting. The quantification of each analysis is shown in the right figure. The experiments were carried out in triplicate (*p < 0.05; **p < 0.01)