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. 2023 Jun 3;15:102. doi: 10.1186/s13195-023-01245-2

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — Plasma Aβ38, Aβ40, and Aβ42 levels in patients with sCAA and controls. Scatter plots in all panels (depicting median and interquartile range). p-values are adjusted for age and sex. A Plasma Aβ38 levels were similar in sCAA patients versus controls (p = 0.092). B Plasma Aβ40 levels were similar in sCAA patients and controls (p = 0.64). C Plasma Aβ42 levels were similar in sCAA patients and controls (p = 0.68). Abbreviations: sCAA, sporadic cerebral amyloid angiopathy Ns, non-significant