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. 2023 Jun 7;14(6):350. doi: 10.1038/s41419-023-05877-y

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — A The effects of the knockdown of UFL1 on tumor growth. Each group contained 8 mice, and data are expressed as mean ± SD. ^*P < 0.05 indicates statistically significant versus negative control. ^**P < 0.01 indicates statistically very significant versus negative control. B The effect of UFL1 knockdown on tumor growth by Ki67 staining. C Examination of RPL10 ufmylation and KLF4 expression in tumor tissues by Co-IP and WB. Tumor tissues were randomly chosen from each group. D The effects of the knockdown of UFL1 on cell stemness related markers by immunochemical staining.