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. 2023 Apr 27;14(17):1606–1617. doi: 10.1111/1759-7714.14906

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© 2023 The Authors. Thoracic Cancer published by China Lung Oncology Group and John Wiley & Sons Australia, Ltd.

This is an open access article under the terms of the http://creativecommons.org/licenses/by-nc/4.0/ License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited and is not used for commercial purposes.

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Upregulated circ_0003028 weakened propofol‐mediated suppression on non‐small cell lung cancer (NSCLC) cell malignant behaviors. (a) Real‐time polymerase chain reaction (RT‐qPCR) analysis of the transfection efficiency of the circ_0003028 overexpression plasmid. (b–h) NSCLC cells were treated as follows: control, propofol, propofol + pCD‐ciR, or propofol + circ_0003028. (b–d) The viability, colony formation, and apoptosis of NSCLC cells were determined. (e and f) Protein levels of cleaved‐caspase‐3 and proliferating cell nuclear antigen (PCNA) in NSCLC cells were detected. (g and h) The migration and invasion of NSCLC cells were evaluated. *p < 0.05.