Figure 3.

Loss of D-La function gives rise to embryonic midgut morphogenesis defects. This allele is balanced over CyO ftz-lacZ which enables the identification of Df(2L)PJ19 homozygotes by the absence of X-gal staining in the central nervous system. The three midgut constrictions that normally occur in wild-type embryos (lines in A) are absent in Df(2L)PJ19 homozygotes (B). Similar results were observed with Df(2L)PJ17 embryos. Embryos were viewed laterally with anterior on the left using a 20× objective and 10× ocular lens.