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. 2023 Feb 7;25(2):301–311. doi: 10.1007/s12017-023-08736-3

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© The Author(s) 2023

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — PGK1 knockdown inhibited OGD/R-induced HAPI microglial cell M1 polarization, inflammation, and glycolysis. A mRNA levels of iNOS and IL-1β, B TNF-α and IL-6 concentrations, C ECAR level, D LDH content, and E, F protein levels of PKM2 and LDHA in HAPI cells transduced with PGK1 shRNA or shNC 24 h after OGD/R. ***p < 0.001 vs. control; ^##p < 0.01, ^###p < 0.001 vs. OGD/R + shNC