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. 2023 Feb 7;25(2):301–311. doi: 10.1007/s12017-023-08736-3

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© The Author(s) 2023

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — PGK1 silencing inhibited microglia M1 polarization, inflammation, and glycolysis in rats with MCAO. A Neurological score, B hematoxylin and eosin staining, C MPO activity, D mRNA levels of iNOS and IL-1β, E TNF-α and IL-6 concentrations, F LDH content, G PGK1 mRNA levels, and H, I PGK1, PKM2, and LDHA protein levels in brain tissues from rats with MCAO. ***p < 0.001 vs. control; ^##p < 0.01, ^###p < 0.001 vs. MCAO + shNC