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. 2023 Jun 15;9:91. doi: 10.1038/s41531-023-00542-9

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — Immunohistochemical analysis of 306C7B3 revealed no labeling of pathological deposits characteristic for other neurodegenerative diseases, including Abeta-positive senile plaques in Alzheimer’s disease (AD, a, b), tau-positive tangles and tufted astrocytes in progressive supranuclear palsy (PSP, c, d), TDP-43-positive inclusions in frontotemporal lobar degeneration with TDP-43 pathology (FTLD-TDP, e, f), or FUS-positive inclusions in frontotemporal lobar degeneration with FET pathology (FTLD-FET, g, h). Immunohistochemistry on formalin fixed paraffin embedded adjacent tissue sections for 306C7B3 (b, d, f, h) and Abeta (a), pTau (c), pTDP-43 (e), or FUS (g). Scale bar 200 μm (a–d), 40 μm (e–h).