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. 2023 Jun 8;3(6):688–704. doi: 10.1038/s43587-023-00431-z

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — a, Plot comparing the adjusted P values of selected inflammation-related GO categories of genes bound by p65 in aged/IgG control, aged/anti-IL-17A/F-treated and adult mice. b, Representative images of genes belonging to inflammation-related GO categories showing binding of p65 to their regulatory areas. c, Plot comparing adjusted P values of selected homeostasis-related GO categories of genes bound by p65 in aged/IgG control, aged/anti-IL-17A/F-treated and adult mice. d, Representative images of genes belonging to the selected epidermal homeostasis-related GO categories showing binding of p65 to their regulatory areas.