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. 2023 Jun 16;3:84. doi: 10.1038/s43856-023-00318-5

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — Longitudinal changes in ALS participants (n = 11, panels in column 1) and controls (n = 13, panels in column 2) in (a) brain FA, (b) brain Fsum, (c) superior temporal cortical thickness, (d) spinal cord CST RD at C2, (e) spinal cord CSA at C2, and (f) brain FDC. The corresponding effect sizes and p-values of the longitudinal change in ALS participants are provided in Table 4 (ALS Amyotrophic lateral sclerosis, FA Fractional anisotropy, Fsum Sum of fiber volume fractions, RD Radial diffusivity, CST Corticospinal tract, C2 Spinal cord C2 level, CSA Cross-sectional area, FDC Fiber density and cross-section).