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. Author manuscript; available in PMC: 2024 Jun 13.
Published in final edited form as: Immunity. 2023 Jun 13;56(6):1303–1319.e5. doi: 10.1016/j.immuni.2023.05.005

Fig. 1. Differentiating antiviral effector CD8+ T cells exhibit highly dynamic accessible chromatin and cBAF occupancy patterns.

Fig. 1.

(A) Signal tracks of ATAC-seq, ARID1A C&R, H3K27ac ChIP-Seq and H3K4me1 ChIP-Seq (GSE89036) in naïve, 48h in vitro activated, or d3, d5, or d8 P14 CD8+ T cells from LCMV-Armstrong infection. (B) ATAC-seq signal coverage and (C) annotation of OCRs from clusters in A. (D-G) Using the OCRs from clusters in (A) we correlated those with ARID1A binding (D), histone modifications to define enhancers (E), predicted TF motif enrichment (F) and actual TF binding (observed/expected) of the indicated TFs from public datasets (GSE54191, GSE192390, GSE166718) (G).