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. 2023 Jun 20;14:3667. doi: 10.1038/s41467-023-39428-y

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© The Author(s) 2023

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — The five CC87 subgroups (ST^WU58-ST^IP186, ST^WU58-ST^IP87-A, ST^WU155-ST^IP21, ST^WU58-ST^IP87-B, ST^WU58-ST^IP24) based on the Warwick University and Institut Pasteur MLST schemes are highlighted in color. The presence of the high pathogenicity island (HPI) is highlighted by red stars and ColV plasmid (defined as proposed by Reid et al.¹⁹) by green triangles. In the outermost circle, colored squares represent the number of mice killed over ten in the mouse model of sepsis¹¹. The 26 ST58 genomes obtained from the study by Reid et al. are in bold. The tree is rooted on IAI1 (non-CC87, phylogroup B1). For the sake of readability, branch lengths are ignored and local support values higher than 0.7 are shown.