The proposed molecular mechanism by which MC-LR interferes with PP1-mediated PI3K/AKT/FOXO1 signaling in follicular granulosa cells to disrupt gonadotropin-dependent follicle maturation and ovulation. Note: 740-YP, a potent PI3K activator; AKT, serine/threonine kinase 1; FSH, follicle-stimulating hormone; FSHR, follicle-stimulating hormone receptor; IGF1, insulin-like growth factor 1; IGF1R, insulin-like growth factor 1 receptor; IRS1, insulin receptor substrate 1; LH, luteinizing hormone; MC-LR, microcystin-LR; OATP, organic anion transporting polypeptide (shown to regulate the cellular uptake of MC-LR); PI3K, phosphatidylinositol 3-kinase; PP1, protein phosphatase 1.